The U.S. government does not require all cattle headed for slaughter to be tested for Mad Cow Disease (i.e. BSE, Bovine Spongioform Encephalopathy), this is a FACT. It is also a FACT that when Europe went to full testing of cattle for slaughter, thousands of infected animals were found. Yet our callous meat industry (you have to be pretty callous to do what they do in the first place) doesn't want to spend the $20 per animal for BSE testing, but spends millions lobbying Congress to maintain the status quo. This is simply not right, and Omaha Steaks is a figurehead in this industry. They CLAIM to only sell high-quality meat, but can that claim be true if the very meat they sell you can easily be tainted with a disease that will make you lose your mind? NOT HARDLY. The purpose of this blog is to pressure the meat industry's figurehead Omaha Steaks to lobby the U.S. government for full testing of every animal headed for slaughter. Anything less is completely irresponsible, even criminal in intent. To think that such crass disregard for humanity could exist in our 'free market' system is not only an indictment of the system but exposes the careless disregard such companies as Omaha Steaks have for their own customers. And if that's how little they care, how can anything else they do or say be trusted?
WHAT MOST PEOPLE DO NOT UNDERSTAND IS THAT MAD COW DISEASE IS FAST BECOMING AN EPIDEMIC IN HUMANS IN AMERICA AND OTHER PARTS OF THE WORLD:
A clinical series with 13% of Alzheimer actually CJD
Manuelidis, Elias E. and Laura Manuelidis
Alzheimer Disease and Associated Disorders_ 3 (1989): 100-109
Suggested Links between Different Types of Dementias: Creutzfeldt-Jakob Disease, Alzheimer Disease, and Retroviral CNS Infections
"In our own neuropathological material, in 46 cases diagnosed clinically as AD [(Alzheimer's)], 6 cases were proven to be CJD at autopsy [13%}."
Peiffer, J. :
Gerstmann-Straussler's disease, atypical multiple sclerosis and carcinomas in a family of sheepbreeders.
Acta Neuropath. 56: 87-92, 1982.
Peiffer (1982) described a family of sheepbreeders in which a father and 2 sons had GSS. All 3 also had congenital hip dysplasia, as did at least 3 other members of the kindred, all females. Atactic symptoms, dysarthria, and personality changes characterized the clinical course of this disorder, which might be labeled atypical multiple sclerosis.
Like CJD , GSS is a form of subacute spongiform encephalopathy. Cases of GSS are clinically similar to the atactic type of CJD. Although there are many neuropathologic similarities, GSS differs from CJD by the presence of kuru-plaques and numerous multicentric, floccular plaques in the cerebral and cerebellar cortex, basal ganglia, and white matter. Whereas only 5 to 15% of CJD cases are familial, most cases of GSS are familial.